A major advantage of immunohistochemistry (IHC) over other immunoassay techniques is that it preserves the architecture of harvested tissue material. This allows researchers to study antigen distribution and relative abundance, which often differ between conditions of health and disease. However, because IHC involves a greater number of protocol steps than many other immunoassay formats, there are more places where things can potentially go wrong. Here, we’ve compiled some common IHC issues and suggested ways of resolving them.

A more complex workflow than other immunoassay techniques

While every immunoassay workflow requires researchers to make certain choices, IHC takes things that much further. As well as deciding between direct or indirect detection, choosing whether to use enzyme-labeled or fluorophore-labeled antibodies, and determining if additional signal amplification is required, researchers performing IHC must also consider whether they will work with frozen or formalin-fixed paraffin-embedded (FFPE) tissue and if epitope retrieval is necessary for antibody reagents to access their targets. Moreover, where FFPE samples are used, the IHC workflow will include steps such as deparaffinization and rehydration—yet more steps requiring careful optimization.

Every protocol step is important

“Each step of the IHC workflow contributes to the final result,” reports Erika Leonard, Director of Quality Control and R&D at Vector Laboratories. “People tend to focus on the primary antibody and the detection reagent, but it is important to make a conscious decision at every point. This includes making sure that every reagent—from the blocking buffer to the mounting media—is compatible with each other as well as the specimen, and optimizing conditions based on the antigen content of your tissue and how your tissue was fixed. For example, some blocking solutions can interfere with primary antibody binding, while the choice of substrate for chromogenic detection can have a significant impact on assay sensitivity. Proper controls are critical throughout to avoid inconsistent or misleading results.”

Multiplexed IHC provides greater context

In recent years, multiplexed IHC has become increasingly popular. According to David Schwartz, Ph.D., CEO and CSO at Cell IDx, this is due to the fact that singleplex staining confirms only that the target of interest is present. “With multiplexed staining, researchers can uncover contextual information of value,” he says. “For example, using multiplexed IHC, it is possible to address questions such as is PD-L1 expressed on tumor cells or immune cells, which specific immune subsets are infiltrating a tumor, and does this information correlate with response to particular therapies?” Historically, multiplexed IHC was limited to just two biomarkers. However, by using Cell IDx’ UltraPlex technology, which is based on cocktails of tag-conjugated primary antibodies that are detected with anti-tag rabbit monoclonals, this is no longer the case.

Troubleshooting tips

Although frozen and FFPE tissue samples involve different IHC staining protocols, both present similar issues. The following table lists some common IHC complaints and potential solutions, with application-specific problems shown in parentheses.

Weak target staining / no target staining

Possible causeSuggested solution
Slides have lost signal during storage

Prepare fresh slides

Store slides at 4°C

Formalin fixation has caused epitope masking

Decrease the fixation time (formalin fixation should not exceed 24 hours)

Select primary antibodies that have been validated for FFPE IHC

Compare different epitope retrieval methods (heat-induced epitope retrieval versus protease-induced epitope retrieval) for unmasking

Samples have not been fully deparaffinized

Increase the deparaffinization time

Prepare fresh dimethylbenzene (xylene) stocks

Samples contain only low concentrations of the target protein

Confirm that an appropriate sample type was used by referring to sites such as UniProt, PAXdb, or proteinatlas.org, and to antibody manufacturers’ datasheets, for information about protein expression

Consider including a signal amplification step

Primary antibodies are not validated for IHC-based detection of native protein

Check that primary antibodies have been validated for the chosen sample type (paraffin sections versus frozen samples) and species

Run a positive control sample, such as a tissue known to endogenously express the target protein or a paraffin-embedded cell pellet, to verify antibody performance

Perform western blots using native and denatured forms of the target protein to confirm the antibody recognizes native protein

Switch to using different primary antibodies

Antibody concentrations are too low

Titrate primary and secondary antibodies to determine optimal concentrations

Use fresh antibodies for each experiment (do not re-use antibodies)

Increase the duration of the primary antibody incubation step

Antibodies have gone bad

Check the expiry dates of antibody reagents

Confirm antibodies have been stored correctly

Avoid freeze-thaw cycles

Antibodies are failing to reach intracellular targets

Optimize the permeabilization step and include the permeabilizing agent in the blocking buffer and antibody diluent

Primary and secondary antibodies are incompatible

Confirm that the correct secondary antibody was used (e.g., if the primary antibody was raised in rabbit, an anti-rabbit secondary is required for detection)

Tissues have dried out

Ensure tissues are covered in liquid throughout the course of the experiment

Enzyme reactions are inhibited

Avoid using sodium azide with horseradish peroxidase (HRP) based detection / phosphate buffer with alkaline phosphatase (AP) based detection

Detection system is not sensitive enough (chromogenic detection)

Switch to a different detection system

Consider performing signal amplification (e.g., using VECTASTAIN® ABC)

Image is under-exposed (fluorescent detection)

Increase the exposure time

 

High background staining

Possible causeSuggested solution
Samples have not been fully deparaffinized

Increase the deparaffinization time

Prepare fresh dimethylbenzene (xylene) stocks

Endogenous peroxidase or phosphatase has not been inactivated

Quench endogenous peroxidase activity using 3% H2O2 in methanol / endogenous phosphatase activity using 1mM Levamisole prior to incubation with primary antibodies

Blocking is insufficient

Optimize the blocking conditions (1 hour blocking with 10% serum from the same host species as the secondary antibody is usually recommended)

Include the blocking agent in antibody diluents

Consider using a glycoprotein-free blocking buffer such as Carbo-Free Blocking Solution

Antibody concentrations are too high

Titrate primary and secondary antibodies to determine optimal concentrations

Reduce the duration of the primary antibody incubation step

Primary antibodies were raised in the same host species as the sample (indirect detection)

Switch to using different primary antibodies

Consider direct detection (with enzyme- or fluorophore-labeled primary antibodies) or try using a biotinylated primary antibody with a conjugated streptavidin detection reagent

Mouse primary antibodies are binding endogenous immunoglobulins in mouse tissue

Include a mouse-on-mouse blocking step (e.g., using a M.O.M.™ Immunodetection Kit)

Secondary antibodies are binding non-specifically to the sample

Use secondary antibodies that have been cross-adsorbed against the host species of the sample

Replace anti-species secondary antibodies with tagged primary antibodies and anti-tag secondaries (e.g., UltraPlex technology from Cell IDx)

Run secondary antibody-only controls

Try pre-incubating secondary antibodies with a blocking solution containing 1-2% serum from the same species as the sample

Endogenous avidin, biotin, or lectins are interfering with avidin/biotin-based detection (avidin is highly-glycosylated and can bind to lectins present in the sample)

Block endogenous avidin/biotin with 0.05% biotin/avidin in PBS, or using a commercial kit (e.g., the Avidin/Biotin Blocking Kit) prior to incubation with primary antibodies

Block endogenous lectins using 0.2 M alpha-methyl mannoside in antibody diluent prior to incubation with primary antibodies

Avoid introducing potential sources of biotin, such as non-fat dry milk or non-IHC grade BSA

Washing is inadequate

Increase the number, volume, and/or duration of wash steps

Substrate was too concentrated or left on for too long (enzymatic detection)

Dilute the substrate

Rinse the substrate off the slides sooner

Tissues have dried out

Ensure tissues are covered in liquid throughout the course of the experiment

Image is over-exposed (fluorescent detection)

Decrease the exposure time

Spectral overlap (fluorescent detection)

Ensure fluorophore-labeled antibodies do not share the same spectral range

 

Autofluorescence (fluorescent detection)

Possible causeSuggested solution
Tissue has inherent autofluorescence

Switch to using infrared fluorophores for detection to minimize overlap

Consider quenching autofluorescence with a product such as the TrueVIEW® Autofluorescence Quenching Kit

Paraffin is generating autofluorescence

Optimize the deparaffinization step

Try using frozen tissue

The fixation method is causing the sample to autofluoresce

Compare different fixation methods

 

Tissue detachment from slide

Possible causeSuggested solution
Sections have not adhered properly

Consider using charged/coated slides (e.g., poly-L-lysine, gelatin, or aminoalkylsilane coated glass) or coat the slides in-house with a reagent such as VECTABOND

Try using a slide warmer after floating sections onto slides

Tissue has not been properly fixed

Optimize the fixation protocol

Slides have been stored incorrectly

Store slides at 4°C

Epitope retrieval is too harsh

Optimize the epitope retrieval method

Bake slides prior to epitope retrieval

Slides have been agitated too vigorously

Use only gentle agitation during incubation and wash steps