Ubr1 (24-Z) Antibody sc-100626 from Santa Cruz Biotechnology, Inc. | Biocompare.com

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Ubr1 (24-Z) Antibody

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Santa Cruz Biotechnology, Inc. for
Ubr1 (24-Z) Antibody

Santa Cruz Biotechnology, Inc.'s Ubr1 (24-Z) Antibody is a Mouse monoclonal antibody. The Ubr1 (24-Z) Antibody was generated using UBR1 as the antigen. It reacts with Homo Sapiens, and Human. This antibody has been shown to work in applications such as: EIA, Immunoassay, Precipitation, ELISA, Immunoprecipitation, and Western Blot.

Description

Ubr1 (24-Z)

Product Specs
ItemUbr1 (24-Z) Antibody
CompanySanta Cruz Biotechnology, Inc.
Price $333.00Supplier Page View Company Product Page
Catalog Numbersc-100626
NCBI Gene AliasesJBS
Antibody TypePrimary Antibodies
Quantity100 µg/ml
Clone24-Z
Reactivityhuman
ApplicationsWB, IP, ELISA
Conjugate/TagUnconjugated
FormatPurified
ClonalityMonoclonal
Hostmouse
NCBI Full Gene Nameubiquitin protein ligase E3 component n-recognin 1
TargetUbr1
IsotypeIgG1

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Santa Cruz Biotechnology, Inc.

Santa Cruz Biotechnology, Inc.

10410 Finnell Street

Dallas, Texas 75220

United States

Phone: 1-800-457-3801
831-457-3801

Company Profile

Website: http://www.scbt.com/

(1) Phosphorylation Impacts N-end Rule Degradation of the Proteolytically Activated Form of BMX KinaseThe Journal of biological chemistryOctober 21, 2016Eldeeb MA, Fahlman RP.

(2) The anti-apoptotic form of tyrosine kinase Lyn that is generated by proteolysis is degraded by the N-end rule pathwayOncotargetMay 15, 2014Eldeeb MA, Fahlman RP.(cat#: F1804) was purchased from Sigma. Rabbit anti-β-actin (I-19, cat#: sc-1616-R), anti-UBR1 (cat#: sc-100626) and anti-UBR2 (cat#: sc-135594) were purchased from Santa Cruz Biotechnology. Secondary antibodies

(3) The anti-apoptotic form of tyrosine kinase Lyn that is generated by proteolysis is degraded by the N-end rule pathwayOncotargetMay 1, 2014Mohamed A. Eldeeb, Richard P. Fahlman

(4) Role of N-end rule ubiquitin ligases UBR1 and UBR2 in regulating the leucine-mTOR signaling pathwayGenes to cells : devoted to molecular & cellular mechanismsJanuary 1, 0001"Kume K, Iizumi Y, Shimada M, Ito Y, Kishi T, Yamaguchi Y, Handa H."

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