Rab3a antibody 107 108 from Synaptic Systems GmbH

Product Specs
ItemRab3a antibody
CompanySynaptic Systems GmbH
Price $420.00Supplier Page View Company Product Page
Catalog Number107 108
Clonalitymonoclonal
Reactivitymouse, rat, human
ApplicationsWB, IP, ICC, IHC
ImmunogenRecombinant protein corresponding to AA 1 to 220 from rat Rab3a
Quantity50 µg
TargetRab3a
Hostrabbit
NCBI Full Gene NameRAB3A, member RAS oncogene family
NCBI Gene AliasesRAB3
Conjugate/TagUnconjugated
ValidationKO
IsotypeIgG1
Formatlyophilized
CloneRb42.2
Antibody TypePrimary antibody

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Synaptic Systems GmbH

Rudolf-Wissell-Str. 28

Goettingen 37079

Germany

Phone: +49-551-50556-0
+49-551-50556-384

Company Profile

Website: http://www.sysy.com/

(1) Research progress in neuroimaging of sporadic early-onset Alzheimer's diseaseFront NeurolApril 10, 2026Xiang Zhang, Chao Yangreplicated and validated. 4.4 SV2A-PET Synaptic loss is also one of the early pathological changes in AD (107, 108), and its correlation with cognitive decline is stronger than that of Aβ or tau burden (109–112). Synaptic

(2) Misfolded Proteins and Cognitive Decline: Mechanistic Insights into Neurodegenerative DisordersNeurol IntMarch 2, 2026Elisa Duranti, Chiara Villapreventing its harmful aggregation while Hsp90 aids in the stabilization of incorrectly folded tau proteins [107,108]. It has been reported that phosphorylation at specific tau residues (Ser202/Thr205) prevents its interaction

(3) Glioblastoma: Overview of Proteomic Investigations and Biobank Approaches for the Development of a Multidisciplinary Translational NetworkCancers (Basel)June 26, 2025Giusy Ciuffreda, Sara Casati, Francesca Brambilla, Mauro Campello, Valentina De Falco, Dario Di Silvestre, Antonio Frigeri, Marco Locatelli, Lorenzo Magrassi, Andrea Salmaggi, et al.stressApoptosis, DNA damage","[82,83,84,85,86,87,88,89,90,91,92,93,94,95,96,97,98,99,100,101,102,103,104,105,106,107,108,109,110,111]" Animal,Tissue,Labeling: iTRAQ,"ILF2, CCT7, CCT4, RPL10A, MSN, PRPS1, TFRC, APEX1","Early

(4) The Neural Palette of Heme: Altered Heme Homeostasis Underlies Defective Neurotransmission, Increased Oxidative Stress, and Disease PathogenesisAntioxidants (Basel)November 22, 2024Adedamola Saidi Soladogun, Li Zhangupregulation during brain injury facilitate neuronal recovery and maturation by mitigating oxidative stress [107,108]. Influence on Axonal Guidance and Synaptic Formation Heme also plays a pivotal role in axonal guidance,

(5) Therapeutic Targets in Innate Immunity to Tackle Alzheimer’s DiseaseCellsAugust 26, 2024Maria L. Serradas, Yingying Ding, Paula V. Martorell, Ida Kulińska, Sergio Castro-GomezAβ","Regulation of microglia survival, activation, and phagocytosis.","Antagonist: 4D9, AL002, CGX101","[106,107,108,109]" Complementsystem,Antibodies,"Synaptic pruning, neuronal migration, polarization and proliferation,

(6) Biomimetic Neuromorphic Sensory System via Electrolyte Gated TransistorsSensors (Basel)July 29, 2024Sheng Li, Lin Gao, Changjian Liu, Haihong Guo, Junsheng YuSynaptic plasticity refers to changes in communication strength based on the synapse’s activity history [107,108]. Paired-pulse facilitation (PPF), a critical form of short-term plasticity (STP), can be characterized

(7) Oxide Ionic Neuro-Transistors for Bio-inspired ComputingNanomaterials (Basel)March 27, 2024Yongli He, Yixin Zhu, Qing Wanartificial neural networks [41,74,98,99,100,101,102,103,104,105,106], and artificial sensory neurons [107,108,109,110,111,112,113,114]. Subsequent chapters describe recent advances in this field. 4. Dynamic Synaptic

(8) Artificial sensory system based on memristive devicesExploration (Beijing)February 1, 2024Ju Young Kwon, Ji Eun Kim, Jong Sung Kim, Suk Yeop Chun, Keunho Soh, Jung Ho YoonSynaptic plasticity is the ability of synaptic connections to change and adapt over time.[ 106 , 107 , 108 ] It is the property that allows synapses to undergo modifications to their strength, efficiency, and

(9) Artificial sensory system based on memristive devicesExploration (Beijing)November 20, 2023Ju Young Kwon, Ji Eun Kim, Jong Sung Kim, Suk Yeop Chun, Keunho Soh, Jung Ho YoonSynaptic plasticity is the ability of synaptic connections to change and adapt over time.[ 106 , 107 , 108 ] It is the property that allows synapses to undergo modifications to their strength, efficiency, and

(10) Septo-hippocampal dynamics and the encoding of space and timeTrends in NeurosciencesSeptember 1, 2023Jennifer C. Robinson, Jacob H. Wilmot, Michael E. Hasselmowhile synaptic currents on a different phase are driven by internal retrieval from within hippocampus [107,108]. Synaptic modification occurs best on the phase of strong external input [108-110]. Without the separation

(11) New Views of the DNA Repair Protein Ataxia–Telangiectasia Mutated in Central Neurons: Contribution in Synaptic Dysfunctions of Neurodevelopmental and Neurodegenerative DiseasesCellsAugust 30, 2023Sabrina Briguglio, Clara Cambria, Elena Albizzati, Elena Marcello, Giovanni Provenzano, Angelisa Frasca, Flavia Antonucci(i) genome stability [107], (ii) neuronal performances, and (iii) survival, as demonstrated in AD [107,108,109]. Contribution of ATM in AD and Synaptic Dysfunctions of AD The involvement of ATM in AD pathogenesis

(12) Axonal energy metabolism, and the effects in aging and neurodegenerative diseasesMolecular NeurodegenerationJuly 20, 2023Sen Yang, Jung Hyun Park, Hui-Chen LuAbeta thread [105]BACE1 accumulates in dystrophic axon [106],"7 M: Glucose metabolism↓ (Female > Male) [107, 108]8 M: Neurotransmitter & glutamine↓ [109]" AD,5XFAD(3 familiar APP and 2 familiar PS1 mutation),Intervention,OSCP

(13) Detecting Early Cognitive Decline in Alzheimer’s Disease with Brain Synaptic Structural and Functional EvaluationBiomedicinesJanuary 26, 2023Samo Ribarič(e.g., epilepsy)) are progressively shifted toward the destruction of functional synapses and neurons [107,108]. 3. Transmission of Brain Action Potentials in Alzheimer’s Disease 3.1. Synaptic Transmission and

(14) Protein Glutathionylation and Glutaredoxin: Role in Neurodegenerative DiseasesAntioxidants (Basel)November 25, 2022Haseena P. A., Latha Diwakar, Vijayalakshmi Ravindranathreactive oxygen species and inactivate glutamine synthetase, which is essential for glutamate metabolism [107,108]. Synaptic dysfunction including loss of dendritic spines is one of the major consequences of AD [109]

(15) Dyrk1a from Gene Function in Development and Physiology to Dosage Correction across Life Span in Down SyndromeGenes (Basel)November 20, 2021Helin Atas-Ozcan, Véronique Brault, Arnaud Duchon, Yann Heraultregulation",[44] RNAPII,RNA polymerase II,Transcription,[105] DNM1,Dynamin 1,Clathrin-mediated vesicle endocytosis,"[106,107,108]" AMPH,Amphiphysin 1,Clathrin-mediated vesicle endocytosis,"[66,107]" SH3GL2,SH3-domain GRB2-like

(16) Molecular Mechanisms of Environmental Metal Neurotoxicity: A Focus on the Interactions of Metals with Synapse Structure and FunctionToxicsAugust 27, 2021Asuncion Carmona, Stéphane Roudeau, Richard Ortegastructure is involved in numerous neurological dysfunctions, including neurodevelopmental disorders [107,108,109], and neurodegenerative diseases such as AD [110,111]. Recent findings from our team suggest that

(17) Tumor Necrosis Factor Alpha in Amyotrophic Lateral Sclerosis: Friend or Foe?CellsMarch 1, 2021Giulia Guidotti, Chiara Scarlata, Liliana Brambilla, Daniela Rossi1054–1059. [CrossRef] Cells 2021, 10, 518 97. 98. 99. 100. 101. 102. 103. 104. 105. 106. 107. 108. 109. 110. 111. 112. 113. 114. 115. 116. 117. 118. 119. 18 of 19 Santello, M.; Bezzi, P.; Volterra,

(18) Regulation of histone deacetylase activities and functions by phosphorylation and its physiological relevanceCellular and Molecular Life SciencesJanuary 1, 2021Sonali Bahl, Edward Setodifferentiation, Synaptic plasticity, Depression, Cocaine-reward behavior","[57,60,85,86,98,99,104,107,108,113,114,127,128,158]" HDAC6,"Neurodegenerative diseases, Mitochondrial transport","[41,129]" HDAC7,"Glioblastoma,

(19) CD8+ T Cell-Mediated Mechanisms Contribute to the Progression of Neurocognitive Impairment in Both Multiple Sclerosis and Alzheimer's Disease?Frontiers in ImmunologyNovember 19, 2020Zorica Stojić-Vukanić, Senka Hadžibegović, Olivier Nicole, Mirjana Nacka-Aleksić, Sanja Leštarević, Gordana LeposavićImmunology | www.frontiersin.org 93. 94. 95. 96. 97. 98. 99. 100. 101. 102. 103. 104. 105. 106. 107. 108. 109. 110. 111. 8 neuroinflammation and synaptic degeneration. Neurol Sci. (2010) 31:S255–9. doi:

(20) Peripheral versus central nervous system APOE in Alzheimer’s disease: Interplay across the blood-brain barrierNeuroscience LettersAugust 1, 2019Dustin Chernick, Stephanie Ortiz-Valle, Angela Jeong, Wenhui Qu, Ling LiFunction The role of APOE and its isoforms in synaptic and cognitive function is well documented [107, 108]. Earlier studies have shown that there are APOE isoform-dependent changes in hippocampal synaptic function

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Rab3a antibody

Rab3a antibody

Rab3a antibody

Rab3a antibody

Rab3a antibody

Rab3a antibody from Synaptic Systems GmbH

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Synaptic Systems GmbH

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Rab3a antibody from Synaptic Systems GmbH

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