Fig 1: The associations between serum CC16 and clinical parameters in CAP patients. The associations between serum CC16 and many clinical parameters were estimated by Spearman correlation coefficient or Pearson rank correlation analyses. The correlations between serum CC16 with white blood cell (WBC), neutrophil, lymphocyte, monocyte, eosinophil, basophil, uric acid, urea nitrogen, creatinine, alanine aminotransferase (ALT), aspartate aminotransferase (AST), creatine kinase (CK), and creatine kinase MB (CKMB) were analyzed by Spearman correlation analysis. In addition, the relationships between serum CC16 with lactate dehydrogenase (LDH), procalcitonin (PCT), tumor necrosis factor alpha (TNF-α), D-Dimer, interleukin 6 (IL-6), and C-reactive protein (CRP) were assessed through Spearman correlation analysis. Red color indicated the positive associations and blue color meant the inverse relationship between serum CC16 and clinical characteristics. The sizes of the circles hinted the strength of the correlations. The larger sizes were, the higher correlations strengths were.
Fig 2: The contents of serum CC16 in CAP patients with different severity. (A–G) The concentration of serum CC16 was evaluated using ELISA. (A) The concentrations of CC16 were compared in cases with different CRB-65 scores. (B) The concentrations of CC16 were compared in cases with different CURB-65 scores. (C) The concentrations of CC16 were compared in cases with different SMART-COP scores. (D) The concentrations of CC16 were compared in cases with different CURXO scores. (E) The concentrations of CC16 were compared in cases with different PSI scores. (F) The concentrations of CC16 were compared in cases with different APACHE II scores. (G) The concentrations of serum CC16 were compared in CAP cases before and after treatment. *P<0.05, **P<0.01.
Fig 3: The contents of serum CC16 in CAP patients with different severity. (A–G) The concentration of serum CC16 was evaluated using ELISA. (A) The concentrations of CC16 were compared in cases with different CRB-65 scores. (B) The concentrations of CC16 were compared in cases with different CURB-65 scores. (C) The concentrations of CC16 were compared in cases with different SMART-COP scores. (D) The concentrations of CC16 were compared in cases with different CURXO scores. (E) The concentrations of CC16 were compared in cases with different PSI scores. (F) The concentrations of CC16 were compared in cases with different APACHE II scores. (G) The concentrations of serum CC16 were compared in CAP cases before and after treatment. *P<0.05, **P<0.01.
Fig 4: The associations between serum CC16 and clinical parameters in CAP patients. The associations between serum CC16 and many clinical parameters were estimated by Spearman correlation coefficient or Pearson rank correlation analyses. The correlations between serum CC16 with white blood cell (WBC), neutrophil, lymphocyte, monocyte, eosinophil, basophil, uric acid, urea nitrogen, creatinine, alanine aminotransferase (ALT), aspartate aminotransferase (AST), creatine kinase (CK), and creatine kinase MB (CKMB) were analyzed by Spearman correlation analysis. In addition, the relationships between serum CC16 with lactate dehydrogenase (LDH), procalcitonin (PCT), tumor necrosis factor alpha (TNF-α), D-Dimer, interleukin 6 (IL-6), and C-reactive protein (CRP) were assessed through Spearman correlation analysis. Red color indicated the positive associations and blue color meant the inverse relationship between serum CC16 and clinical characteristics. The sizes of the circles hinted the strength of the correlations. The larger sizes were, the higher correlations strengths were.
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