Anti-mouse IL-4 mAb (11B11), azide free 3311-3N-500 from Mabtech AB | Biocompare.com

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Anti-mouse IL-4 mAb (11B11), azide free

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Anti-mouse IL-4 mAb (11B11), azide free

Mabtech AB's Anti-mouse IL-4 mAb (11B11), azide free is a Rat monoclonal antibody. The Anti-mouse IL-4 mAb (11B11), azide free Antibody was generated using IL4, and Interleukin 4 as the antigen. It reacts with Mouse. This antibody has been shown to work in applications such as: Block/Neutralize/Inhibit.

Description

This monoclonal antibody is recommended for neutralization of mouse IL-4 bioactivity

Product Specs
ItemAnti-mouse IL-4 mAb (11B11), azide free
CompanyMabtech AB
Price Pricing Info Supplier Page View Company Product Page
Catalog Number3311-3N-500
Quantity500 µg
ApplicationsNeutralization
Conjugate/Tagunconjugated
Formatunconjugated
Reactivitymouse
ClonalityMonoclonal
IsotypeIgG1
Clone11B11
HostRat
TargetIL-4
NCBI Full Gene Nameinterleukin 4
NCBI Gene AliasesBSF-1, Il-4
Components11B11, purified, supplied at 1 mg/ml in PBS free of preservatives
Puritypurified
Concentration1 mg/ml

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Mabtech AB

Mabtech AB

Box 1233

Nacka Strand, SE-131 28

Sweden

Phone: +46 8 716 27 00
+46 8 716 27 01

Company Profile

Website: http://www.mabtech.com

(1) BALB/c mice infected with antimony treatment refractory isolate of Leishmania braziliensis present severe lesions due to IL-4 productionPLoS neglected tropical diseasesMarch 1, 2011Costa DL, Carregaro V, Lima-Júnior DS, Silva NM, Milanezi CM, Cardoso CR, Giudice Â, de Jesus AR, Carvalho EM, Almeida RP, et al.

(2) Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosisPloS oneJanuary 1, 2011Cavassani KA, Tristao FS, Oliveira LL, Rocha FA, Vancim JO, Moreira AP, Campanelli AP, Panagio LA, Milanezi CM, Martinez R, et al.

(3) Interleukin (IL)-4 promotes T helper type 2-biased natural killer T (NKT) cell expansion, which is regulated by NKT cell-derived interferon-gamma and IL-4ImmunologyJanuary 1, 2008Iizuka A, Ikarashi Y, Yoshida M, Heike Y, Takeda K, Quinn G, Wakasugi H, Kitagawa M, Takaue Y.

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