RABBIT ANTI GAP43

Published customer image:
Rabbit anti GAP43 antibody (AHP669) used to demonstrate GAP43 expression in nerve fibers in a rat myocardial ischemia model by immunohistochemistry on formalin fixed paraffin embedded tissue sections.
Image caption:
GAP43 immunohistochemical staining of the infarct border zone (DAB color, ×200). (a) Sham-operated group. The distribution of GAP43-positive nerve fibers is orderly. (b) Control ischemic group. GAP43-positive nerve fibers in ventricular muscle increased significantly, it is also thickened and disordered. (c) Preischemic treatment group. (d) Delayed application group. The distribution of GAP43-positive nerve fibers after treatment with desipramine significantly improved.

From: Wu X, Jiang H, Yu L, Hu X, Liu W.
Desipramine pretreatment improves sympathetic remodeling and ventricular fibrillation threshold after myocardial ischemia.
.
This is from an open access article distributed under the terms of the.

Published customer image:
Rabbit anti GAP43 antibody (AHP669) used to demonstrate GAP43 expression in nerve fibers in a rat myocardial ischemia model by immunohistochemistry on formalin fixed paraffin embedded tissue sections.
Image caption:
GAP43 immunohistochemical staining of the infarct border zone (DAB color, ×200). (a) Sham-operated group. The distribution of GAP43-positive nerve fibers is orderly. (b) Control ischemic group. GAP43-positive nerve fibers in ventricular muscle increased significantly, it is also thickened and disordered. (c) Preischemic treatment group. (d) Delayed application group. The distribution of GAP43-positive nerve fibers after treatment with desipramine significantly improved.

From: Wu X, Jiang H, Yu L, Hu X, Liu W.
Desipramine pretreatment improves sympathetic remodeling and ventricular fibrillation threshold after myocardial ischemia.
J Biomed Biotechnol. 2012;2012:732909.
This is from an open access article distributed under the terms of the Creative Commons Attribution License.