Ly-49I Antibody, anti-mouse, Biotin 130-106-107 from Miltenyi Biotec | Biocompare.com

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Ly-49I Antibody, anti-mouse, Biotin

The Ly-49I Antibody, anti-mouse, Biotin from Miltenyi Biotec is a Mouse Monoclonal antibody to Killer Cell Lectin-like Receptor Subfamily A, Member 9, and KLRA9. This antibody recognizes Mouse antigen. The Ly-49I Antibody, anti-mouse, Biotin has been validated for the following applications: Flow Cytometry.

Product Specs
ItemLy-49I Antibody, anti-mouse, Biotin
CompanyMiltenyi Biotec
Price Pricing Info Supplier Page View Company Product Page
Catalog Number130-106-107
Quantity30 µg in 1 mL
ApplicationsFlow cytometry
Conjugate/TagBiotin
FormatReagents are supplied in buffer containing stabilizer and 0.05% sodium azide
Reactivitymouse
TargetLy-49I
IsotypeIgG1
CloneYLI-90
Hostmouse
Antibody TypePrimary antibodies
Clonalitymonoclonal
Regulatory StatusFor research use only
Concentration1:10
NCBI Full Gene Namekiller cell lectin-like receptor subfamily A, member 9
NCBI Gene AliasesLY49, LY49I1, Ly4, Ly49I
Research AreaFor research use only

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Miltenyi Biotec

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Miltenyi Biotec

Miltenyi Biotec Inc.

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Fax: +49 2204 8306-235

Email: macs@miltenyibiotec.de

Company Profile

Website: www.miltenyibiotec.com

(1) Ly49 family receptors are required for cancer immunosurveillance mediated by natural killer cellsCancer researchJuly 15, 2014Tu MM, Mahmoud AB, Wight A, Mottashed A, Bélanger S, Rahim MM, Abou-Samra E, Makrigiannis AP.

(2) NKG2D receptor-mediated NK cell function is regulated by inhibitory Ly49 receptorsBloodJanuary 1, 2005Regunathan J, Chen Y, Wang D, Malarkannan S.

(3) Crystal structure of the Ly49I natural killer cell receptor reveals variability in dimerization mode within the Ly49 familyJournal of molecular biologyJuly 12, 2002Dimasi N, Sawicki MW, Reineck LA, Li Y, Natarajan K, Margulies DH, Mariuzza RA.

(4) Ly-49 multigene family. New members of a superfamily of type II membrane proteins with lectin-like domainsJournal of immunology (Baltimore, Md. : 1950)August 15, 1991Wong S, Freeman JD, Kelleher C, Mager D, Takei F.

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