Biotin anti-mouse CD252 (OX40 Ligand) 108803 from BioLegend | Biocompare.com

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Biotin anti-mouse CD252 (OX40 Ligand)

BioLegend's Biotin anti-mouse CD252 (OX40 Ligand) is a Rat monoclonal antibody. The Biotin anti-mouse CD252 (OX40 Ligand) Antibody was generated using TNFSF4 as the antigen. It reacts with Mouse. This antibody has been shown to work in applications such as: Flow Cytometry.

Description

Biotin anti-mouse CD252 (OX40 Ligand) [RM134L]

Product Specs
ItemBiotin anti-mouse CD252 (OX40 Ligand)
CompanyBioLegend
Price Pricing Info Supplier Page View Company Product Page
Catalog Number108803
NCBI Gene AliasesAth-1, Ath1, CD134L, OX-40L, Ox40l, TXGP1, Tnlg2b, Txgp1l, gp34
Quantity50 µg
Conjugate/TagBIOTIN
IsotypeIgG2b, Kappa
HostRat
ApplicationsFlow Cytometry (FC)
ReactivityMouse
CloneRM134L
Antibody TypePrimary
NCBI Full Gene Nametumor necrosis factor (ligand) superfamily, member 4
Concentration0.50 mg/mL
FormatPhosphate-buffered solution, pH 7.2, containing 0.09% sodium azide
ImmunogenRat NRK-52E cell line transfected with mouse OX-40L
ClonalityMonoclonal
TargetCD252

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BioLegend

BioLegend

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San Diego, California 92121

United States

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Website: biolegend.com

(1) Conserved and Differential Features of TNF Superfamily Ligand Expression on APC Subsets over the Course of a Chronic Viral Infection in MiceImmunoHorizonsDecember 20, 2018Wang KC, Chu KL, Batista NV, Watts TH.

(2) Dichotomous Expression of TNF Superfamily Ligands on Antigen-Presenting Cells Controls Post-priming Anti-viral CD4⁺ T Cell ImmunityImmunityNovember 21, 2017Chang YH, Wang KC, Chu KL, Clouthier DL, Tran AT, Torres Perez MS, Zhou AC, Abdul-Sater AA, Watts TH.

(3) TSLP and downstream molecules in experimental mouse allergic conjunctivitisInvestigative ophthalmology & visual scienceJune 1, 2010Zheng X, Ma P, de Paiva CS, Cunningham MA, Hwang CS, Pflugfelder SC, Li DQ.

(4) [Determination of gamma-glutamyltransferase in the differential diagnosis of jaundice: value in the ALT/GGT index]Revista do Hospital das ClínicasOctober 1, 1978L C da Silva, A Sáez-Alquézar, E P Camargo, A da Rocha Campos, H S Júnior, A de Oliveira e Silva, N Ohtsuki, S Raia

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