Neurodegenerative diseases resulting from progressive loss of neuronal function is an active area of research. Alzheimer’s disease, affecting over 29 million people worldwide as of 2015, implicates a causative role of amyloid beta aggregates within the brain. These are formed from the peptide cleavage products of the amyloid beta precursor protein (APP). Parkinson’s disease affects as many as 6.2 million people, causing abnormal motor functions and cognitive degeneration with age. While there is no clear genetic link to PD, the synuclein alpha (SNCA) protein is noted for its role in the formation of Lewy bodies in Parkinson’s patients. Huntington’s disease is a rarer, inheritable disease, known to be caused by a mutant gene coding for huntingtin (HTT). HTT is highly expressed in brain and, when mutated, can form nonfunctional aggregates. Other proteins reported to be involved in neurodegeneration include parkin, SOD1, GSK3, presenilin 1, and tau.